I have to answer this question for school, and although I’ve looked everywhere, I cannot find a good answer.

Diabetes can be caused by an autoimmune process that destroys insulin producing cells (type I) or through a variety of mutations and or excessive weight which prevents the normal function of insulin following meals.
Symptoms vary depending on the severity of the disease.
See: http://www.cdc.gov/diabetes/faq/basics.htm
Although a blood test is mroe definitive, excess glucose in urine is a sign of diabetes.
Nephritis or glomerular nephritis resulting from diabetes, or diabetic nephropathy, can be diagnosed by excess levels of protein and or blood in urine. It is believes that excess glucose in the blood increases the rate of flow of blood into the kidney. This strains the filtration machinery and has the effect of also raising blood pressure.

Tags : , , ,

File under : Diabetes Mellitus

Permalink : http://diabetes-type-1.org/diabetes-mellitus/identify-the-cause-of-diabetes-mellitus-and-nephritis-its-physical-smptoms-how-it-is-detected-by-urinalysis
Trackback : http://diabetes-type-1.org/diabetes-mellitus/identify-the-cause-of-diabetes-mellitus-and-nephritis-its-physical-smptoms-how-it-is-detected-by-urinalysis/trackback

One Response

  1. Professor M

    Diabetes can be caused by an autoimmune process that destroys insulin producing cells (type I) or through a variety of mutations and or excessive weight which prevents the normal function of insulin following meals.
    Symptoms vary depending on the severity of the disease.
    See: http://www.cdc.gov/diabetes/faq/basics.htm
    Although a blood test is mroe definitive, excess glucose in urine is a sign of diabetes.
    Nephritis or glomerular nephritis resulting from diabetes, or diabetic nephropathy, can be diagnosed by excess levels of protein and or blood in urine. It is believes that excess glucose in the blood increases the rate of flow of blood into the kidney. This strains the filtration machinery and has the effect of also raising blood pressure.
    References :

Leave a Reply